Skip to main content
Fig. 4 | BMC Neuroscience

Fig. 4

From: KDM4A, involved in the inflammatory and oxidative stress caused by traumatic brain injury-hemorrhagic shock, partly through the regulation of the microglia M1 polarization

Fig. 4

KDM4A involved in the inflammation and oxidative stress in LPS-induced BV2 cells. (A). The mRNA and protein expression levels of KDM4A in KDM4A down-regulated BV2 cells (n = 3). (B). The mRNA and protein expression levels of KDM4A in KDM4A down-regulated LPS-induced BV2 cells (n = 3). (C-F). The content of TNF-α, IL-1β, MDA and GSH in KDM4A inhibited LPS-induced BV2 cells were measured by detection kits (n = 3). ANOVA and Tukey’s multiple comparisons test were used to analyze data. (G). ROS level in KDM4A down-regulated LPS-induced BV2 cells. Data were collected from three independent experiments and expressed as mean ± SD. Full-length blots are presented in Supplementary Figure S3

Back to article page