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Fig. 5 | BMC Neuroscience

Fig. 5

From: KDM4A, involved in the inflammatory and oxidative stress caused by traumatic brain injury-hemorrhagic shock, partly through the regulation of the microglia M1 polarization

Fig. 5

Suppressed KDM4A abolished microglia M1 polarization in LPS-induced BV2 cells. (A-B). The mRNA and protein expression levels of iNOS and COX-2 in KDM4A down-regulated BV2 cells (n = 3). ANOVA and Tukey’s multiple comparisons test were used to analyze data. (C). Images of double immunofluorescent staining for KDM4A (red) and CD16/32 (green) in KDM4A inhibited LPS-induced BV2 cells. Blue fluorescence represented DAPI. Data were collected from three independent experiments and expressed as mean ± SD. Full-length blots are presented in Supplementary Figure S4

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BMC Neuroscience

ISSN: 1471-2202

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