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Fig. 5 | BMC Neuroscience

Fig. 5

From: Sodium formononetin-3'-sulphonate alleviates cerebral ischemia–reperfusion injury in rats via suppressing endoplasmic reticulum stress-mediated apoptosis

Fig. 5

Schematic diagram of the molecular mechanisms underlying the protective effects of Sul-F treatment against cerebral I/R injury. Sul-F treatment alleviated the I/R-induced activation of ER stress, thereby inhibiting multiple downstream pathways, including PERK/eIF2α/ATF4, IRE1/Caspase12/Caspase3 and CHOP/Bcl-2/Bax signal pathway

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BMC Neuroscience

ISSN: 1471-2202

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