Fig. 4
From: TNFAIP1 contributes to the neurotoxicity induced by Aβ25–35 in Neuro2a cells
Effect of Aβ25–35 and TNFAIP1 on p-Akt, p-CREB and Bcl-2 expression. a p-Akt, p-CREB and Bcl-2 protein levels were measured by Western blot in N2a cells cultured in 6 wells plate and treated with dose-dependent Aβ25–35 (0–20 μM). b p-Akt, p-CREB and Bcl-2 protein levels were measured by Western blot in N2a cells cultured in 6 wells plate and transfected with Myc-TNFAIP1 and pCMV-Myc (0–4 μg). c Quantitative analysis of p-CREB protein levels (fold of control). Data are expressed as mean ± SD, n = 3, *p < 0.05, **p < 0.01 compared with Myc-TNFAIP1 (0 μg). d Quantitative analysis of p-Akt protein levels (fold of control). Data are expressed as mean ± SD, n = 3, *p < 0.05 compared with Myc-TNFAIP1 (0 μg). e Quantitative analysis of Bcl-2 protein levels (fold of control). Data are expressed as mean ± SD, n = 3, *p < 0.05 compared with Myc-TNFAIP1 (0 μg)