Fig. 5From: Higher intrinsic network excitability in ventral compared with the dorsal hippocampus is controlled less effectively by GABAB receptorsDependence of spfps-nmda on NMDARs and glutamate release. Example recordings illustrating that spfps-nmda were completely abolished by the competitive antagonist of NMDARs CPP (a) or the open channel blocker of the same receptors MK-801 (b). Spfps-nmda were also blocked by activation of adenosine A1 receptors by their agonist 2-chloroadenosine (c). Data shown in “b” and “c” were obtained from two DH slicesBack to article page