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Table 1 Patients' clinical characteristics.

From: The longitudinal changes of BOLD response and cerebral hemodynamics from acute to subacute stroke. A fMRI and TCD study

Pt

Age

AH

Clinical

Symptoms

NIHSS

Exp 0

Lesion site

Stenosis ICA (%)

Vessel

Occlusion

Trombolysis

Risk factors

      

AH

UH

   

1, ♂

66

L

Aphasia

8

PC

0

0

MCA-branch

Y

Hypert, smoke

2, ♂

69

L

Aphasia/Hemiparesis FB

14

FC, PC

0

0

MCA-branch

Y

AF

3, ♂

63

L

Aphasia

6

FC, PC

0

0

MCA-branch

Y

Hypert, AF

4, ♂

46

L

Aphasia

6

PC

100

0

M2

N

Hypert, smoke

5, ♀

72

L

Aphasia

3

TC

0

0

MCA-branch

Y

Hypert, Chol

6, ♀

44

L

Aphasia

7

FC

0

0

MCA-branch

Y

Hypert

  1. Pt: patient; ♂: man, ♀: woman; AH: affected hemisphere, UH: unaffected hemisphere; L = left, R = right; Clinical Symptoms: FBC = facial/brachial/crural; NIHSS Exp 0: NIH stroke scale at emergency room admission; Lesion site: BG = basal ganglia, IC = internal capsula, PC = parietal cortex, FC = frontal cortex, TC = Temporal Cortex; Stenosis ICA = internal carotid artery; Vessel Occlusion: MCA = Middle Cerebral Artery, M2 = M2 segment of middle cerebral artery, Trombolysis: Y = yes, N = no; Risk factors: hypert = Hypertension, AF = atrial fibrillation, Smoke = cigarette smoking, Chol = hypercholesterolemia.